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22 It is speculated that fibrin deposition with thrombus formation on the damaged vessel wall occurs through modification of the electrostatic charge of the endothelial cells
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Recent research determined that chronic alcohol intake inhibited the activation of mammalian target of rapamycin complex 1 (mTORC1), leading to an uncoupling status between lipolysis and the browning/thermogenesis of white adipose tissue (WAT), and thus exacerbating hepatic lipid accumulation, inflammation, and fibrosis in the development and progression of ALD (Song et al., 2023)
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