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The loss of a major hepatic form of GST, which represents over 3% of total soluble protein (Fountoulakis et al ., 2000), would have been expected to predispose the animals to hepa-totoxicity through a reduction in the glutathione conju-gation of NAPQI (Coles et al ., 1988)
161 There is extensive cross-regulation between autophagy and ER stress, 162 and SIRT1 may coordinate the feedback mechanisms between these processes
Surely the HRV would recover as I adapted
I.El-KarefA.ElsherbinyN
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