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Physicians should carefully monitor these clinical effects related to #GLP1 usage
Uric acid induces cardiomyocyte apoptosis via activation of calpain-1 and endoplasmic reticulum stress
However, with levothyroxine, you are really replacing a deficiency, so once you are stabilized and in a 'normal range' for your thyroid hormone, it's not much to be concerned about
This genetic context does not determine medication response outright, but it may help contextualize individual tolerability patterns
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