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Generally, alveolar macrophage inflammation and epithelial cell damage are the triggering events of pulmonary fibrosis, followed by fibroblast proliferation and myofibroblast activation in response to cytokines, which in turn results in excessive extracellular matrix (ECM) deposition [2, 3]
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Elevated calcium levels can significantly impact cardiac function, and by adhering to the highest safety standards, we prevent such risks
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