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Product inhibition and energy state also controls the TCA flux
, diabetes, dysbiosis), chemical reactivity enables spontaneous modification, potentiated by glyoxalase system dysregulation: methylglyoxal is converted by glyoxalase 1 (GLO1) to lactoylglutathione (LGSH), which undergoes non-enzymatic acyl transfer to lysine residues (55)
Overgaard RV, Delff PH, Petri KCC, et al
(*) Hiu qu dng glutathione mi ngi l khc nhau
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