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Current Pharmaceutical Design , 20(7), 11211125
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Exercise as a Secondary Metabolic Stressor Adipose tissue dysfunction in obesity promotes the release of small extracellular vesicles enriched with oxidatively stressed mitochondrial components [66, 91], reflecting unresolved mitochondrial strain
These designs have been useful in dissecting which weight-related endpoints depend on IGF-1 and which are truly GH-independent a question of ongoing interest in obesity and metabolic science
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