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Spectrum of GLP-1 Effects in Parkinsons Disease Clinical Improvement GLP-1 RAs have emerged as promising therapeutic candidates in PD, particularly due to their dual action on metabolic and neurodegenerative pathways
NTS catecholamine neurons mediate hypoglycemic hunger via medial hypothalamic feeding pathways
[Studies on anti-ulcer effects of a new compound, zinc L-carnosine (Z-103)]
These effects are also seen in patients with T2DM and in other insulin-resistant states such as obesity [135, 136] and may be mediated in part by restoration of vascular insulin sensitivity [137]
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