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CONCLUSIONS GLP-1 RAs and multi-agonist therapies combining GLP-1 with GIP and/or GCG agonism embody transformative therapeutic approaches for MASLD/MASH, addressing multiple pathogenic pathways through complementary mechanisms of action
[16] These receptors, when activated by allulose, may trigger signalling cascades that promote incretin hormone release, though the magnitude and clinical significance of this effect in humans requires further investigation
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