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Signs of vascular dysfunction were completely prevented by benfotiamine administration, and biomarkers of endothelial dysfunction and oxidative stress were significantly reduced (Stirban 2006)
These effects are attributed to indirect modulation of signaling pathways involved in cell survival rather than direct stimulation of neuronal activity
The choice usually comes down to whether KPV's anti-inflammatory mechanism is part of the research interest
Thumbnail: Gary Brecka by Gage Skidmore , CC BY-SA 2.0 FAQs What peptides does Gary Brecka point to for injury recovery
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