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We use the model to study the extent of liver damage caused by overdoses or by chronic use of therapeutic doses, and the effects of polymorphisms in glucoronidation enzymes
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The model was first fitted to the training concentrations (37.5 and 25 M doxorubicin), and then used to simulate the test concentrations (50, 12.5 and 10 M doxorubicin) as a means of blind validation, showing good accordance between the experimental and in silico outputs for all concentrations of doxorubicin
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Similarly, in WAT, a direct comparison between Gipr fl/fl vehicle and Gipr Syn/ vehicle groups resulted in 1,856 DEGs with 909 upregulated and 947 downregulated genes
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