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Methyl-metabolite depletion elicits adaptive responses to support heterochromatin stability and epigenetic persistence
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CrossRef Clinical Characteristics of Early-Stage Gallbladder Cancer Bonggyu Seong, Ju Yeun Song, Sun Youn Bae, Kwang Hyuck Lee, Jong Kyun Lee, Jong Chul Rhee, Kyu Taek Lee Korean Journal of Medicine.2015
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This suppression is independent of plasma cholesterol changes and BW reduction through regulating markers of plaque instability and inflammation including those linked to plaque hemorrhage (CD163), matrix turnover (matrix metalloproteinase (MMP)-3, MMP-13), cholesterol metabolism (prostaglandin I2 synthase (PTGIS), ATP-binding cassette transporter 1 (ABCA1)), leukocyte recruitment (C-C motif chemokine ligand 2 (CCL2), osteopontin (OPN), interleukin (IL)-6), and leukocyte rolling, extravasation and adhesion (vascular cell adhesion molecule (VCAM)-1, E-selectin (SELE)) [16]
