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The protein expression and activation of FAK and paxillin were determined by Western blot analysis, and the phosphorylation levels of both FAK and paxillin were dose dependently increased by BPC 157 while the total amounts of protein was unaltered

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The three components are paired in a single product because their mechanisms are commonly framed as complementary in research and clinical-anecdote contexts: GHK-Cu modulates collagen and elastin synthesis, antioxidant gene expression, and dermal extracellular matrix remodeling