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High expression of HNF4 in EC may supplement intracellular GSH through a non-xCT-dependent pathway by activating metabolic enzymes involved in methionine metabolism, which may represent a key mechanism underlying ferroptosis resistance in EC
Cureus, 13 (12), e20828
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Therapeutic use of oral BPC-157 focuses on comprehensive gut health optimization, treatment of various digestive disorders, support for leaky gut syndrome, and systemic healing enhancement
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