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Altered functioning of the glyoxalase enzyme system, with associated accumulation of glycation products, has been shown as another possible pathogenic mechanism in NAFLD (41, 42)
Jastreboff AM, Aronne LJ, Ahmad NN, et al
G., Liu, S., Sun, C
The pathological results showed no observed damage to the liver, kidney and other important organs of mice, and the treatment effect of ATC tumor was significant
363 In hypoxic tumors, antagonizing HIF-1 or its transcriptional targets (e.g., PDK1/3, MT2A) may prevent mitochondrial copper sequestration and promote lethal copper accumulation
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