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Nonetheless, our study demonstrates that a persistent imbalance between oxidative stress toxicity and reduced ANTIOX is a key component of Long COVID and that increased aldehyde, neutrophil-associated MPO, protein carbonyl, and NO production are the major neurotoxic pathways, which, when combined with decreased ANTIOX, may have a variety of detrimental effects as reviewed before [81]

The result: increased systemic oxidative stress that can amplify fatigue, brain fog, and metabolic inflammation even as weight drops

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Furthermore, tolerability issues require effective gastrointestinal side effect mitigation, as these are a primary reason for cessation

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