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Transitioning GLP-1 treatments to the oral route would, therefore, represent a major advancement
Both medications mimic GLP-1, a type of gut hormone that plays a role in regulating appetite, blood sugar, and satiety (how satisfied you feel after eating)
We must move beyond viewing neuropathy as a vague "side effect." It is a specific, mechanistic outcome of NAD depletion and mitochondrial failure
A., Hagins, H., et al
By targeting multiple metabolic pathways, these dual incretin therapies may produce stronger metabolic improvements than traditional GLP-1 medications alone
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