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This gene, subsequently named spxR (pyruvate oxidase regulator), encodes a regulator capable of activating the expression of spxB and, consequently, the endogenous production of H 2 O 2 in response to the metabolic state (Ramos-Montanez et al., 2008

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L., Langenberg, P., Meyers, D

121,122 Ji et al found that oral administration of TMAO significantly exacerbated demyelination in spontaneously hypertensive rats, with the underlying mechanism possibly related to TMAO-induced pyroptosis of oligodendrocytes and inflammation