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Whilst GLP-1 receptor activation can exert multiple direct neuroprotective effects, the protective influence of GLP-1 receptor agonists may occur indirectly through the restoration of the dysfunctional insulin signaling associated with neurodegenerative disease

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In addition, a number of drugs are being developed that target both the GIP and GLP-1 pathways, and triple-agonists at the glucagon, GIP, and GLP-1 receptors

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