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Though absent in AIM SRCR2, Ca 2+ site 1 is also likely to be present in AIM SRCR1 based on sequence conservation

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Kaufman, D

5 Summary and outlook Heat stress-induced spermatogenic dysfunction in males is not caused by a single form of heat damage but rather results from the synergistic activity of multiple pathways, including protein homeostasis imbalance and oxidative stress accumulation, which collectively impair germ cells and disrupt spermatogenesis

In women, OS is implicated in various reproductive disorders, including polycystic ovary syndrome, endometriosis, and premature ovarian failure, leading to diminished oocyte quality, disrupted folliculogenesis, and poorer reproductive outcomes