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The accelerated vascular growth into the bone defect site occurs through the CGRP/FAK/VEGF signaling axis
While BPC-157 activates the FAK-paxillin pathway for fibroblast movement and promotes angiogenesis via VEGFR2-Akt-eNOS signaling, TB-500 binds G-actin to facilitate cell migration and promotes angiogenesis through VEGF/HIF-1alpha pathways
This process assists the body in repairing a variety of tissues, including those of the stomach, intestines, skin, vasculature, nerve cells, and the brain
Journal of molecular medicine (Berlin, Germany), 95(3), 323333
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