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The peptide contains an N-terminal acetylation that differs from the naturally occurring form

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Drawing upon the known property of exosomes to targetedly deliver bioactive molecules, we raise a speculative hypothesis: exosomes released by damaged endothelium might have the theoretical capacity to induce secondary cuproptosis responses in the vascular microenvironment, potentially by encapsulating abnormal copper transporters or cytotoxic lipoylated protein aggregates

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