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Liraglutide treatment reduced the proportion of the -1 subcluster, further highlighting the protective role of liraglutide (Figure 3F)
This spatial difference (matrix vs
Sepsis-induced endothelial-derived exosomes are often accompanied by high expression of tissue factor and adhesion of citrullinated histone H3, molecular features that reveal the malignant interaction between endothelial injury and neutrophil extracellular traps (NETs) (105, 108)
These drugs target metabolic pathways involving glucose regulation and appetite control, not the hormonal and inflammatory mechanisms underlying endometriosis
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