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doi: 10.1038/s41574-019-0176-8 2
That choice addresses the symptom but not the mechanism and it introduces a new problem
While regulation of iron metabolism-related proteins or the use of iron chelators can reduce intracellular labile Fe 2+ levels and inhibit ferroptosis, current chelators lack tissue and cellular specificity and may interfere with neurophysiological functions, thereby limiting their clinical applicability
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