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Collectively, these results validate TMT-TAILs as an effective strategy for off-target substrate nomination and suggest that X(PC) induces a multipathway cell death response by cleaving endogenous caspase targets across both apoptotic and inflammatory pathways
This is precisely why the distinction matters here: the material listed on this page is the short-acting no-DAC form, so this paper's durability results do not describe it
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