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Chronic inflammation, often driven by persistent infections, autoimmune diseases, or environmental factors, can create a pro-tumorigenic environment [110]
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The residue-specific reactivities and k red values for C151 and C208 in roGFP2(SSG) as well as active-site residue C29 in reduced dithiol PfGrx C88S and monothiol PfGrx C32S/C88S in Fig
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