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Activation of these receptors triggers two complementary processes: it accelerates lipolysis (the breakdown of stored triglycerides into free fatty acids for energy) while simultaneously inhibiting lipogenesis (the conversion of glucose and other substrates into new fat stores)

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Smith AC, Mears AJ, Bunker R, Ahmed A, MacKenzie M, Schwartzentruber JA, Beaulieu CL, Ferretti E, Majewski J, Bulman DE, et al

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[10] [22] Disease-modifying therapies commonly used in MS include: Injectable therapies : Interferon beta preparations (Avonex, Rebif, Betaseron) and glatiramer acetate (Copaxone) Oral agents : Fingolimod (Gilenya), dimethyl fumarate (Tecfidera), teriflunomide (Aubagio), siponimod (Mayzent), ozanimod (Zeposia), and cladribine (Mavenclad) Infusion therapies : Natalizumab (Tysabri), ocrelizumab (Ocrevus), alemtuzumab (Lemtrada) Subcutaneous injection : Ofatumumab (Kesimpta) No direct drug-drug interactions between tirzepatide and these DMTs have been identified in pharmacological studies