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Additionally, BPC-157 influences FAK-Src-EGFR growth factor transactivation, which supports cellular signaling cascades involved in repair and regeneration

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Presented for research literature review only Hrehorck M et al

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A particular relationship was established with the NO-system, and the advantage of the BPC 157 over the corresponding standard agents (i.e., corticosteroids, sulfasalazine, H2 blockers, anticholinergics, and proton pump inhibitors) which showed only weak, if any, effect on these fistulas closing (Klicek et al., 2008