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Increasing evidence suggests that microbial communities residing within tumors or at distant mucosal sites can drive resistance to anticancer therapies through diverse mechanisms, including modulation of drug metabolism and transport, induction of ROS, impairment of DDR, activation of oncogenic signaling, and suppression of antitumor immunity

This is one of the reasons published Tirzepatide research is typically discussed not only in terms of efficacy, but also in terms of tolerability during escalation , GI event burden and the balance between metabolic benefit and dropout risk

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