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Since BCAAs are essential amino acids that cannot be synthesised endogenously, sustained pharmacological enhancement of BCAA catabolism carries theoretical risks of reducing BCAA availability below levels required for muscle protein synthesis, neurological function, and other critical physiological processes

[4] It appears to further lack the proposed implications sometimes seen with hGH supplementation, such as long-bone growth, reduced sensitivity to insulin, and edema

Reference List Cleveland Clinic

Previous clinical trials have demonstrated that glucagon-like peptide-1 receptor agonists (GLP-1 RAs) improve cardiovascular outcomes, including heart failure, as well as all-cause death 5,6,7,8

IGF-1 is associated with muscle repair, cellular growth, and metabolic signaling