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Depletion of intracellular glutathione contributes to JNK-mediated death receptor 5 upregulation and apoptosis induction by the novel synthetic triterpenoid methyl-2-cyano-3, 12-dioxooleana-1, 9-dien-28-oate (CDDO-Me)

Our results of the EF% (EF%: SMD: -4.13, 95% CI -5.56, -2.69, p = 0.00001) and FS% (FS%: SMD: -2.91, 95% CI -3.96, -1.86, p = 0.00001) are in agreement with having decreased values in mice induced with MI, as opposed to the mice treated with FGF-9 after inducing MI, which displayed an improvement in the left ventricular output measured by the elevation of the EF% and the FS% as parameters of cardiomyopathy function compared to the non-diabetic controls

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