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Conclusively, bearing in mind that insulinotropic or incretinotropic effects are not secondary to any single receptor modulation, whereby pools of different luminal stimuli act synergistically on tens of different chemosensors during their intestinal transit and absorption, while interacting with the microflora metabolism, rendering the restoration of a healthy physiology in diabetic patients with the pharmacological correction of a single axis, highly improbable
Therefore, further targeted in vivo and human-relevant mechanistic studies are required to determine whether incretin receptor agonism can meaningfully modify fibrotic progression in pulmonary disease
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