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For instance, in NSCLC, treatment with first- and second-generation EGFR tyrosine kinase inhibitors (TKIs) often leads to the development of the EGFR T790M mutation, while third-generation inhibitors subsequently select for the C797S mutation, both of which confer resistance
Confirm identity is supported by LC-MS or mass spectrometry, plus copper-complex confirmation
marneffei shows sensitivity to various oxidative stressors, whether glutathione metabolic gene expression is dependent on aftA was not tested 65
It can be a powerful tool in the right clinical context
Malins et al
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