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Gut barrier dysfunction is common in Long COVID, ME/CFS, and MCAS, driving systemic inflammation

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Here we find increased proportion of pro-inflammatory and mesenchymal fibroblast subpopulations and senescent fibroblasts, and enhanced expression of senescence-associated secretory phenotype genes using single-cell RNA sequencing analysis, as well as elevated p16 protein and more -galactosidase-positive cells in keloids

We discuss emerging CGM metrics, diagnostic challenges, and critical evidence gaps that must be addressed to define its role in preventive care