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The pathophysiology of doxorubicin includes inhibition of DNA/RNA/protein synthesis, increased production of ROS, impaired energy metabolism, mitochondrial dysfunction, cardiomyocytes apoptosis, interstitial fibrosis, autophagy disorders and disorders of intracellular calcium homeostasis (Kong et al

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Specifically, targeting oxidative stress through molecular pathways such as MAPK, TLR4/NF-B, Nrf2, and PI3K/Akt could offer new therapeutic avenues for IBD management

Multiple animal studies show that BPC-157 can help with healing injuries to the myocardial tissue, such as injury from a heart attack

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