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SM102 treatment enhanced tear secretion, prolonged tear film breakup time, reversed corneal tissue damage, and increased conjunctival goblet cell density

using GLP-1s could reach 25 million by 2030 (rising from 10 million in 2025)

The accumulation of these compounds primarily results from defects in D-2-hydroxyglutarate dehydrogenase or L-2-hydroxyglutarate dehydrogenase or due to defects in both enzymes

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