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[7] In a 2026 neuropathic-pain study, photoaffinity-probe experiments identified LanCL1 as a binding target of LAT8881-derived probe material in nervous-system models, and LanCL1 knockdown blocked the reported LAT8881 activity in those models
This comparison helps clarify how AOD 9604 is positioned relative to upstream GH-axis modulators in experimental systems
Then, weekly for a few more weeks
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