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Glutathione might inhibit the effects of cerebral infarction and enhance antiapoptotic signaling after ischemic stroke, suggesting that GSH may be a potent therapeutic antioxidant that can attenuate severe pathologies after ischemic stroke, and stimulating GSH synthesis through administration of GSH precursors and micronutrients like selenium can optimize GSH and GSH peroxidase for optimal antioxidant defense in cerebral ischemia (155, 156)
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UA activates Nrf2 and leads to the upregulation of GCLM gene expression, boosting GSH synthesis and release in astrocytes, which in turn protects glial cells and neighboring neurons [177]
Read the full review Nutrition & Metabolic Deficiencies and metabolic drivers of cognitive symptoms
Nevertheless, beneficial treatments include correcting acidosis, blood transfusion, and antioxidant supplementation (70
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