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Notably, ME treatment largely restored transport velocity under these compromised conditions, effectively mitigating the loss of Band 3mediated anion exchange ( Figure 5A

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The expression of the VD metabolism genes CYP2R1 and CYP27A1 (25-hydroxylase), CYP27B1 (1--hydroxylase), and vitamin D receptor (VDR) were downregulated in the livers of mice fed an HFD (GSH- deficient) compared with control diet-fed group

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