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Demonstrates increased resistance to enzymatic degradation in plasma compared with native GHRH due to its trans3hexenoic Nterminal group, resulting in a longer halflife and more sustained GH axis activation
Pericardial fat, visceral abdominal fat, cardiovascular disease risk factors, and vascular calcification in a community-based sample: the Framingham Heart Study
10.1186/s12906-024-04491-5 124 LiangY.XuM
In parallel, it alters transcriptional pathways involved in angiogenesis, cytokine production, and tissue remodeling, including PI3K/Akt and NF-B cascades
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