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These physiologically low-toxicity ligands, upon AhR activation, tend to induce transcriptional programs that maintain mucosal homeostasis and immune tolerance, including stabilization of Treg phenotypes, enhancement of the IL-22-mediated barrier protection axis, and suppression of excessive Th17-driven inflammation, thereby functionally biasing AhR signaling toward immune protection in this context (Zelante et al., 2013
Follow your prescribers advice and avoid binge episodes
Nunziata, J
Their studies measured outcomes at 12 weeks
Author contributions JW designed the research, JW and RWcontributed to the literature database search, data collection, data extraction, data analysis, and writing of the manuscript
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