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A second family, BPC-157 and TB-500, targets tissue repair rather than the GH axis: BPC-157 is proposed to work through angiogenesis, nitric-oxide signaling, and anti-inflammatory activity, while TB-500 binds actin to promote cell migration, blood-vessel formation, and collagen deposition
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More studies are needed to explore whether the GPX4-mediated activation of parthanatos is a widespread mechanism in other disease models, and how the lipid peroxidation involved in the regulation of parthanatos
