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This approach reduces formulation complexity and cost, while also contributing to the development of products with improved purity and health profiles

GHK-Cu acts locally and systemically on extracellular matrix remodeling, wound healing, and inflammatory regulation

This dual effect enhances endurance capacity and supports favorable body composition changes by promoting the preferential use of stored fat for fuel

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Mechanistically, pterostilbene treatment of RBL-2H3 cells increased phosphorylation of Serine/Threonine Kinase 11 (LKB1), AMPK, and acetyl co-A carboxylase, and decreased proteins involved in FcRI-mediated signaling, including phosphorylated p38, ERK, JNK, IKK and mTOR, suggesting pterostilbene both activates the LKB1/AMPK pathway and inhibits FcRI signaling [209]