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doi: 10.1038/nature04330 74

Collectively, these findings indicate that reduced adipocyte mitochondrial CI activity during obesity is a key driver of visceral WAT metabolic dysfunction and subsequent systemic impairment in metabolic health

I'm guessing my doctor will go along with trying this, all arrows seem to point in that direction in terms of causality, and there's no real downside even if it doesn't do the trick

Ferroptosis is characterized by the depletion of intracellular GSH and decreased activity of GPx4, which in turn results in the accumulation of lipid peroxides and Fe(II)-catalyzed oxidation of lipids by the Fenton reaction and increased ROS formation

From the point of view of industrial development, postbiotics, which are the metabolic byproducts of probiotics, are nonliving and, thus, more stable and have a longer shelf life than live probiotic strains