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These phosphorylated R-SMADs dissociate from the receptor and form a trimeric complex with SMAD4, which translocates into the nucleus to regulate transcription of fibrosis-related genes, including -smooth muscle actin (-SMA) and collagen I [40, 41]

The uncropped and unprocessed scans are provided in the Source Data file

M.et al (2002)

Studies in mice confirmed that animals lacking beta-3 receptors showed no response to AOD-9604, proving this pathway is essential to the mechanism

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