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Additionally, GLP-1 can inhibit apoptosis by activating the PI3K/Akt pathway, promoting the anti-apoptotic protein Bcl-2 and hindering the activity of caspases
This observation raises questions about the underlying mechanisms of senescence and the necessity of addressing the root causes that drive cellular senescence, rather than merely focusing on the symptomatic removal of senescent cells
Just that we have very limited data on the systemic effects
The drug also significantly increased dopamine, catalase activity, and glutathione content in the brain
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