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By blocking TREK-1, PE-22-28: Increases neuronal excitability : Reduces potassium efflux, depolarizing neurons toward firing threshold Enhances serotonergic transmission : TREK-1 is expressed on serotonergic neurons in the dorsal raphe nucleus
We here discover the further biocompatibility of ZIL based on the molecular mechanisms and suggest ZIL as a functionalised solution superior to DMSO, serving as a cryoprotectant and a vehicle for hydrophobic drugs, for instance
Generally, the pharmacokinetic parameters have a good agreement between experimental and simulated curves
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