Mechanistically, because acylcarnitines are direct metabolites derived from fatty acid and organic acid catabolism, disruptions in systemic FAO under metabolic stress, such as during the introduction of enteral feeding in a compromised gut, could theoretically contribute to gut-specific toxicity
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In diabetes, HFpEF is predominantly driven by coronary microvascular disease/dysfunction, systemic inflammation, as well as cardiovascular autonomic dysfunction (which is very specific to diabetes) and a deficient heart oxidative metabolism, all leading to remodeling of the heart and increased stiffness, while HFrEF is predominantly driven by adverse remodeling secondary to accelerated atherosclerotic CVD (ASCVD)