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This substantially reduces the frequency of administration and side effects [58]

We speculated that GSH may inhibit high glucose-induced PSC activation via the ROS/TGF/SMAD pathway, thereby inhibiting fibrosis progression

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Observations indicating a parallel, opposing influence of Nrf2 on the transcriptional upregulation of pro-inflammatory genes suggest that targeted pharmacological activation of the Nrf2 pathway could help in the controlled reduction of oxidative stress (Kobayashi et al

A researcher leaves a vial on the lab bench, thinking its stable, only to find their next experiment yields bizarre, unreliable data