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Indeed, nine patients that were originally reported as LCAD-deficient, diagnosed by a reduction in palmitoyl-CoA degradation [7], were reported to have normal LCAD protein levels on western blot [8] and upon subsequent analyses many were in fact VLCAD-deficient [9]

The paper established MOTS-c as an "exercise mimetic" and launched the field of mitochondrial-derived peptide research (Lee C et al., Cell Metab

When administered at the recommended dosage, the risk of side effects is minimal

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