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in ex vivo distal-lung fibroblasts from smokers and COPD donors, GHK reorganized the actin cytoskeleton, elevated integrin 1, and restored collagen-I contraction and remodeling capacity to levels comparable with TGF- treatment
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Because copper is a required cofactor for numerous enzymes involved in cellular metabolism and antioxidant activity, researchers studying GHK-Cus copper-binding properties often position it as a tool for understanding copper-dependent signaling pathways generally, not just those specific to skin or tissue repair
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